Ohri, Sujata Saraswat
Attenuating the endoplasmic reticulum stress response improves functional recovery after spinal cord injury. [electronic resource]
- Glia Oct 2011
- 1489-502 p. digital
Publication Type: Journal Article; Research Support, N.I.H., Extramural; Research Support, Non-U.S. Gov't
ISSN: 1098-1136
Standard No.: 10.1002/glia.21191 doi
Subjects--Topical Terms: Activating Transcription Factor 4--metabolism Analysis of Variance Animals Apoptosis--genetics Basic Helix-Loop-Helix Transcription Factors--genetics Caspase 3--genetics DNA-Binding Proteins--metabolism Disease Models, Animal Endoplasmic Reticulum Stress--genetics Eukaryotic Initiation Factor-2--metabolism Female Locomotion--physiology Mice Mice, Inbred C57BL Mice, Knockout Myelin Basic Protein--genetics Nerve Fibers, Myelinated--metabolism Nerve Tissue Proteins--genetics Oligodendrocyte Transcription Factor 2 Oligodendroglia--metabolism RNA, Messenger--metabolism Recovery of Function--physiology Regulatory Factor X Transcription Factors Spinal Cord Injuries--metabolism Time Factors Transcription Factor CHOP--deficiency Transcription Factors--metabolism Unfolded Protein Response--genetics Up-Regulation--genetics