Weinger, Jason G <br/><br/>
Loss of the receptor tyrosine kinase Axl leads to enhanced inflammation in the CNS and delayed removal of myelin debris during experimental autoimmune encephalomyelitis.  [electronic resource] 

 -  Journal of neuroinflammation  May 2011 
 - 49 p.  digital 
<br/><br/> Publication Type: Journal Article; Research Support, N.I.H., Extramural; Research Support, Non-U.S. Gov't 
<br/><br/><label>ISSN: </label>1742-2094 
<br/><br/><label>Standard No.: </label>10.1186/1742-2094-8-49  doi 
<br/><br/><label>Subjects--Topical Terms: </label><br/>Animals<br/>Central Nervous System--immunology<br/>Cytokines--immunology<br/>Encephalomyelitis, Autoimmune, Experimental--immunology<br/>Glycoproteins--immunology<br/>Inflammation--immunology<br/>Macrophages--cytology<br/>Mice<br/>Mice, Knockout<br/>Microglia--cytology<br/>Myelin Sheath--metabolism<br/>Myelin-Oligodendrocyte Glycoprotein<br/>Peptide Fragments--immunology<br/>Proto-Oncogene Proteins--genetics<br/>Receptor Protein-Tyrosine Kinases--genetics<br/>Axl Receptor Tyrosine Kinase