Zhang, Qing-Jun <br/><br/>
The histone trimethyllysine demethylase JMJD2A promotes cardiac hypertrophy in response to hypertrophic stimuli in mice.  [electronic resource] 

 -  The Journal of clinical investigation  Jun 2011 
 - 2447-56 p.  digital 
<br/><br/> Publication Type: Journal Article; Research Support, N.I.H., Extramural; Research Support, Non-U.S. Gov't 
<br/><br/><label>ISSN: </label>1558-8238 
<br/><br/><label>Standard No.: </label>10.1172/JCI46277  doi 
<br/><br/><label>Subjects--Topical Terms: </label><br/>Animals<br/>Aortic Diseases--complications<br/>Base Sequence<br/>Cells, Cultured--drug effects<br/>Constriction, Pathologic--complications<br/>Female<br/>Gene Expression Regulation<br/>Heart Failure--enzymology<br/>Histone Demethylases--deficiency<br/>Histones--metabolism<br/>Humans<br/>Hypertrophy, Left Ventricular--enzymology<br/>Intracellular Signaling Peptides and Proteins<br/>Jumonji Domain-Containing Histone Demethylases--genetics<br/>LIM Domain Proteins<br/>Male<br/>Methylation<br/>Mice<br/>Mice, Inbred C57BL<br/>Mice, Transgenic<br/>Molecular Sequence Data<br/>Muscle Proteins--biosynthesis<br/>Myocytes, Cardiac--drug effects<br/>Nuclear Proteins--physiology<br/>Promoter Regions, Genetic<br/>Protein Processing, Post-Translational<br/>RNA, Small Interfering--pharmacology<br/>Rats<br/>Serum Response Element<br/>Trans-Activators--physiology<br/>Ventricular Remodeling--genetics