Aging-induced collateral dysfunction: impaired responsiveness of collaterals and susceptibility to apoptosis via dysfunctional eNOS signaling. [electronic resource]
- Journal of cardiovascular translational research Dec 2011
- 779-89 p. digital
Publication Type: Journal Article; Research Support, N.I.H., Extramural
1937-5395
10.1007/s12265-011-9280-4 doi
Age Factors Aging--metabolism Animals Aorta--enzymology Apoptosis Arterial Occlusive Diseases--enzymology Blotting, Western Bone Marrow Transplantation Cell Movement Cells, Cultured Collateral Circulation Cyclic Nucleotide Phosphodiesterases, Type 5--metabolism Disease Models, Animal Endothelial Cells--enzymology Female Femoral Artery--surgery Laser-Doppler Flowmetry Ligation Lower Extremity--blood supply Mice Mice, Inbred C57BL Muscle, Smooth, Vascular--enzymology Myocytes, Smooth Muscle--enzymology Neovascularization, Physiologic Nitric Oxide Synthase Type III--metabolism Perfusion Imaging--methods Phosphorylation Regional Blood Flow Signal Transduction