Peng, Cheng <br/><br/>
The response to TLR ligation of human CD16⁺CD14⁻ monocytes is weakly modulated as a consequence of persistent infection with the hepatitis C virus.  [electronic resource] 

 -  Molecular immunology  Jul 2011 
 - 1505-11 p.  digital 
<br/><br/> Publication Type: Journal Article 
<br/><br/><label>ISSN: </label>1872-9142 
<br/><br/><label>Standard No.: </label>10.1016/j.molimm.2011.04.008  doi 
<br/><br/><label>Subjects--Topical Terms: </label><br/>Adult<br/>Cell Count<br/>Chemokine CCL4--biosynthesis<br/>Cytokines--biosynthesis<br/>Female<br/>Hepacivirus--immunology<br/>Hepatitis C, Chronic--immunology<br/>Humans<br/>Imidazoles--pharmacology<br/>Lipopolysaccharide Receptors--immunology<br/>Lipopolysaccharides--immunology<br/>Male<br/>Middle Aged<br/>Monocytes--immunology<br/>Receptors, IgG--immunology<br/>Toll-Like Receptor 4--metabolism<br/>Toll-Like Receptor 8--agonists<br/>Tumor Necrosis Factor-alpha--biosynthesis