Jakob, Burkhard <br/><br/>
DNA double-strand breaks in heterochromatin elicit fast repair protein recruitment, histone H2AX phosphorylation and relocation to euchromatin.  [electronic resource] 

 -  Nucleic acids research  Aug 2011 
 - 6489-99 p.  digital 
<br/><br/> Publication Type: Journal Article; Research Support, Non-U.S. Gov't 
<br/><br/><label>ISSN: </label>1362-4962 
<br/><br/><label>Standard No.: </label>10.1093/nar/gkr230  doi 
<br/><br/><label>Subjects--Topical Terms: </label><br/>Animals<br/>Ataxia Telangiectasia Mutated Proteins<br/>Cell Cycle Proteins--metabolism<br/>Cells, Cultured<br/>Chromobox Protein Homolog 5<br/>Chromosomal Proteins, Non-Histone--metabolism<br/>DNA Breaks, Double-Stranded<br/>DNA Repair<br/>DNA-Binding Proteins--metabolism<br/>Euchromatin--metabolism<br/>HeLa Cells<br/>Heterochromatin--metabolism<br/>Histones--metabolism<br/>Humans<br/>Kinetics<br/>Mice<br/>Phosphorylation<br/>Protein Serine-Threonine Kinases--metabolism<br/>Tumor Suppressor Proteins--metabolism<br/>X-ray Repair Cross Complementing Protein 1