Xiao, Lin <br/><br/>
Switching of N-methyl-D-aspartate (NMDA) receptor-favorite intracellular signal pathways from ERK1/2 protein to p38 mitogen-activated protein kinase leads to developmental changes in NMDA neurotoxicity.  [electronic resource] 

 -  The Journal of biological chemistry  Jun 2011 
 - 20175-93 p.  digital 
<br/><br/> Publication Type: Journal Article; Research Support, Non-U.S. Gov't 
<br/><br/><label>ISSN: </label>1083-351X 
<br/><br/><label>Standard No.: </label>10.1074/jbc.M110.188854  doi 
<br/><br/><label>Subjects--Topical Terms: </label><br/>Aging--physiology<br/>Animals<br/>Calcium--metabolism<br/>Cell Differentiation--physiology<br/>Embryo, Mammalian--metabolism<br/>Enzyme Activation--physiology<br/>Hippocampus--cytology<br/>MAP Kinase Signaling System--physiology<br/>Male<br/>Mitogen-Activated Protein Kinase 3--genetics<br/>N-Methylaspartate--genetics<br/>Neurons--cytology<br/>Rats<br/>Rats, Sprague-Dawley<br/>Receptors, N-Methyl-D-Aspartate--genetics<br/>p38 Mitogen-Activated Protein Kinases--genetics