Lee, M-H <br/><br/>
TGF-β induces TIAF1 self-aggregation via type II receptor-independent signaling that leads to generation of amyloid β plaques in Alzheimer's disease.  [electronic resource] 

 -  Cell death & disease  Dec 2010 
 - e110 p.  digital 
<br/><br/> Publication Type: Journal Article; Research Support, Non-U.S. Gov't 
<br/><br/><label>ISSN: </label>2041-4889 
<br/><br/><label>Standard No.: </label>10.1038/cddis.2010.83  doi 
<br/><br/><label>Subjects--Topical Terms: </label><br/>Alzheimer Disease--genetics<br/>Amyloid beta-Peptides--metabolism<br/>Amyloid beta-Protein Precursor--genetics<br/>Animals<br/>Apoptosis<br/>Apoptosis Regulatory Proteins--genetics<br/>COS Cells<br/>Chlorocebus aethiops<br/>Hippocampus--metabolism<br/>Humans<br/>Mice<br/>Mice, Transgenic<br/>Nuclear Proteins--genetics<br/>Phosphorylation<br/>Plaque, Amyloid--etiology<br/>Polymerization<br/>Signal Transduction<br/>Smad4 Protein--analysis<br/>Transforming Growth Factor beta--metabolism<br/>Two-Hybrid System Techniques