Yowtak, June <br/><br/>
Reactive oxygen species contribute to neuropathic pain by reducing spinal GABA release.  [electronic resource] 

 -  Pain  Apr 2011 
 - 844-852 p.  digital 
<br/><br/> Publication Type: Journal Article; Research Support, N.I.H., Extramural 
<br/><br/><label>ISSN: </label>1872-6623 
<br/><br/><label>Standard No.: </label>10.1016/j.pain.2010.12.034  doi 
<br/><br/><label>Subjects--Topical Terms: </label><br/>Analysis of Variance<br/>Animals<br/>Bicuculline--pharmacology<br/>Cyclic N-Oxides--pharmacology<br/>Disease Models, Animal<br/>Dose-Response Relationship, Drug<br/>Excitatory Postsynaptic Potentials--drug effects<br/>Free Radical Scavengers--pharmacology<br/>GABA-A Receptor Antagonists--pharmacology<br/>Ganglia, Spinal--pathology<br/>Hyperalgesia--physiopathology<br/>Injections, Spinal--methods<br/>Male<br/>Mice<br/>Neuralgia--drug therapy<br/>Neurons--drug effects<br/>Reactive Oxygen Species--metabolism<br/>Spinal Cord--metabolism<br/>Spinal Nerves--metabolism<br/>Time Factors<br/>gamma-Aminobutyric Acid--metabolism<br/>tert-Butylhydroperoxide--pharmacology