Wallis, Susan K <br/><br/>
A polymorphism in the interleukin-4 receptor affects the ability of interleukin-4 to regulate Th17 cells: a possible immunoregulatory mechanism for genetic control of the severity of rheumatoid arthritis.  [electronic resource] 

 -  Arthritis research & therapy  Feb 2011 
 - R15 p.  digital 
<br/><br/> Publication Type: Journal Article; Research Support, N.I.H., Extramural; Research Support, Non-U.S. Gov't 
<br/><br/><label>ISSN: </label>1478-6362 
<br/><br/><label>Standard No.: </label>10.1186/ar3239  doi 
<br/><br/><label>Subjects--Topical Terms: </label><br/>Adult<br/>Arthritis, Rheumatoid--genetics<br/>Cell Separation<br/>Cells, Cultured<br/>Cross-Sectional Studies<br/>Female<br/>Genetic Predisposition to Disease--genetics<br/>Genotype<br/>Humans<br/>Interleukin-17--biosynthesis<br/>Interleukin-4--immunology<br/>Male<br/>Middle Aged<br/>Pilot Projects<br/>Polymorphism, Single Nucleotide<br/>Receptors, Interleukin-4--genetics<br/>Signal Transduction--genetics<br/>Th17 Cells--immunology<br/>Young Adult