Jia, Fang <br/><br/>
AMP-activated protein kinase inhibits homocysteine-induced dysfunction and apoptosis in endothelial progenitor cells.  [electronic resource] 

 -  Cardiovascular drugs and therapy  Feb 2011 
 - 21-9 p.  digital 
<br/><br/> Publication Type: Journal Article; Research Support, Non-U.S. Gov't 
<br/><br/><label>ISSN: </label>1573-7241 
<br/><br/><label>Standard No.: </label>10.1007/s10557-010-6277-1  doi 
<br/><br/><label>Subjects--Topical Terms: </label><br/>AMP-Activated Protein Kinases--metabolism<br/>Aminoimidazole Carboxamide--analogs & derivatives<br/>Apoptosis--drug effects<br/>Atorvastatin<br/>Cell Movement--drug effects<br/>Cell Proliferation--drug effects<br/>Cells, Cultured<br/>Down-Regulation--drug effects<br/>Endothelial Cells--cytology<br/>Heptanoic Acids--pharmacology<br/>Homocysteine--antagonists & inhibitors<br/>Humans<br/>NADPH Oxidase 4<br/>NADPH Oxidases--metabolism<br/>Nitric Oxide--metabolism<br/>Nitric Oxide Synthase Type III--genetics<br/>Phosphorylation--drug effects<br/>Pyrroles--pharmacology<br/>Reactive Oxygen Species--metabolism<br/>Ribonucleotides--pharmacology<br/>Stem Cells--cytology<br/>Up-Regulation--drug effects