Crowley, Steven D <br/><br/>
Comments on Point:Counterpoint: The dominant contributor to systemic hypertension: Chronic activation of the sympathetic nervous system vs. Activation of the intrarenal renin-angiotensin system. AT1 receptors in the kidney mediate the chronic hypertensive response to angiotensin II.  [electronic resource] 

 -  Journal of applied physiology (Bethesda, Md. : 1985)  12 2010 
 - 2012-3 p.  digital 
<br/><br/> Publication Type: Journal Article; Comment 
<br/><br/><label>ISSN: </label>1522-1601 
<br/><br/><label>Standard No.: </label>10.1152/japplphysiol.01160.2010  doi 
<br/><br/><label>Subjects--Topical Terms: </label><br/>Angiotensin II--metabolism<br/>Angiotensin II Type 1 Receptor Blockers--therapeutic use<br/>Animals<br/>Antihypertensive Agents--therapeutic use<br/>Blood Pressure--drug effects<br/>Humans<br/>Hypertension--drug therapy<br/>Kidney--drug effects<br/>Mice<br/>Mice, Knockout<br/>Receptor, Angiotensin, Type 1--deficiency<br/>Renin-Angiotensin System--drug effects<br/>Sympathetic Nervous System--metabolism<br/>Treatment Outcome