Lee, Sang-Min <br/><br/>
Stimulation of FasL induces production of proinflammatory mediators through activation of mitogen-activated protein kinases and nuclear factor-κB in THP-1 cells.  [electronic resource] 

 -  Inflammation  Feb 2012 
 - 1-10 p.  digital 
<br/><br/> Publication Type: Journal Article; Research Support, Non-U.S. Gov't 
<br/><br/><label>ISSN: </label>1573-2576 
<br/><br/><label>Standard No.: </label>10.1007/s10753-010-9283-3  doi 
<br/><br/><label>Subjects--Topical Terms: </label><br/>Antibodies, Monoclonal<br/>Antigens, Surface--immunology<br/>Cell Line, Tumor<br/>Extracellular Signal-Regulated MAP Kinases--metabolism<br/>Fas Ligand Protein--genetics<br/>Humans<br/>Interleukin-8--biosynthesis<br/>JNK Mitogen-Activated Protein Kinases--metabolism<br/>MAP Kinase Signaling System<br/>Macrophage Activation<br/>Macrophages--immunology<br/>Matrix Metalloproteinase 9--biosynthesis<br/>Membrane Glycoproteins--immunology<br/>NF-kappa B--metabolism<br/>RNA Interference<br/>RNA, Small Interfering<br/>Receptors, Fc--genetics<br/>Recombinant Fusion Proteins--pharmacology<br/>Tumor Necrosis Factor-alpha--biosynthesis<br/>fas Receptor--genetics<br/>p38 Mitogen-Activated Protein Kinases--metabolism