Huang, Lianghu <br/><br/>
Increased susceptibility to acute kidney injury due to endoplasmic reticulum stress in mice lacking tumor necrosis factor-α and its receptor 1.  [electronic resource] 

 -  Kidney international  Mar 2011 
 - 613-623 p.  digital 
<br/><br/> Publication Type: Journal Article; Research Support, N.I.H., Extramural 
<br/><br/><label>ISSN: </label>1523-1755 
<br/><br/><label>Standard No.: </label>10.1038/ki.2010.469  doi 
<br/><br/><label>Subjects--Topical Terms: </label><br/>Acute Kidney Injury--chemically induced<br/>Animals<br/>Apoptosis<br/>Cells, Cultured<br/>Disease Models, Animal<br/>Disease Susceptibility<br/>Endoplasmic Reticulum--drug effects<br/>Enzyme Inhibitors--pharmacology<br/>Eukaryotic Initiation Factor-2--metabolism<br/>Female<br/>Kidney Tubules, Proximal--drug effects<br/>Lipid Metabolism<br/>Mice<br/>Mice, Inbred C57BL<br/>Mice, Knockout<br/>Phosphoprotein Phosphatases--antagonists & inhibitors<br/>Phosphorylation<br/>Receptors, Tumor Necrosis Factor, Type I--deficiency<br/>Receptors, Tumor Necrosis Factor, Type II--deficiency<br/>Severity of Illness Index<br/>Signal Transduction<br/>Time Factors<br/>Tumor Necrosis Factor-alpha--deficiency<br/>Tunicamycin