Ma, Chi <br/><br/>
Neuroprotection by histone deacetylase-7 (HDAC7) occurs by inhibition of c-jun expression through a deacetylase-independent mechanism.  [electronic resource] 

 -  The Journal of biological chemistry  Feb 2011 
 - 4819-28 p.  digital 
<br/><br/> Publication Type: Journal Article; Research Support, N.I.H., Extramural 
<br/><br/><label>ISSN: </label>1083-351X 
<br/><br/><label>Standard No.: </label>10.1074/jbc.M110.146860  doi 
<br/><br/><label>Subjects--Topical Terms: </label><br/>Animals<br/>Apoptosis--drug effects<br/>Cell Line, Tumor<br/>Cerebellum--cytology<br/>Extracellular Signal-Regulated MAP Kinases--antagonists & inhibitors<br/>Female<br/>Gene Expression Regulation--drug effects<br/>Histone Deacetylase Inhibitors--pharmacology<br/>Histone Deacetylases--genetics<br/>MAP Kinase Kinase Kinases--antagonists & inhibitors<br/>MAP Kinase Signaling System--drug effects<br/>Male<br/>Mice<br/>Mice, Transgenic<br/>Neurons--cytology<br/>Phosphatidylinositol 3-Kinases--metabolism<br/>Phosphoinositide-3 Kinase Inhibitors<br/>Protein Kinase Inhibitors--pharmacology<br/>Protein Structure, Tertiary<br/>Proto-Oncogene Proteins c-akt--antagonists & inhibitors<br/>Proto-Oncogene Proteins c-jun--biosynthesis<br/>Rats<br/>Rats, Wistar<br/>Transcription, Genetic--drug effects