Chen, Shmuel <br/><br/>
An increase in persistent sodium current contributes to intrinsic neuronal bursting after status epilepticus.  [electronic resource] 

 -  Journal of neurophysiology  Jan 2011 
 - 117-29 p.  digital 
<br/><br/> Publication Type: Journal Article; Research Support, Non-U.S. Gov't 
<br/><br/><label>ISSN: </label>1522-1598 
<br/><br/><label>Standard No.: </label>10.1152/jn.00184.2010  doi 
<br/><br/><label>Subjects--Topical Terms: </label><br/>Animals<br/>CA1 Region, Hippocampal--cytology<br/>Calcium Channel Blockers--pharmacology<br/>Calcium Channels, T-Type--drug effects<br/>Male<br/>Models, Animal<br/>Neurons--drug effects<br/>Patch-Clamp Techniques<br/>Pilocarpine--adverse effects<br/>Rats<br/>Rats, Inbred Strains<br/>Rats, Wistar<br/>Sodium Channel Blockers--pharmacology<br/>Sodium Channels--drug effects<br/>Status Epilepticus--chemically induced<br/>Tetrodotoxin--pharmacology<br/>Time Factors