Symons, J David <br/><br/>
Knockout of insulin receptors in cardiomyocytes attenuates coronary arterial dysfunction induced by pressure overload.  [electronic resource] 

 -  American journal of physiology. Heart and circulatory physiology  Jan 2011 
 - H374-81 p.  digital 
<br/><br/> Publication Type: Journal Article; Research Support, N.I.H., Extramural; Research Support, Non-U.S. Gov't 
<br/><br/><label>ISSN: </label>1522-1539 
<br/><br/><label>Standard No.: </label>10.1152/ajpheart.01200.2009  doi 
<br/><br/><label>Subjects--Topical Terms: </label><br/>Acetylcholine--pharmacology<br/>Analysis of Variance<br/>Animals<br/>Blotting, Western<br/>Coronary Vessels--drug effects<br/>Echocardiography<br/>Genotype<br/>Hypertrophy, Left Ventricular--genetics<br/>Male<br/>Mice<br/>Mice, Knockout<br/>Myocytes, Cardiac--drug effects<br/>Nitroprusside--pharmacology<br/>Receptor, Insulin--genetics<br/>Reverse Transcriptase Polymerase Chain Reaction<br/>Ventricular Dysfunction, Left--genetics<br/>omega-N-Methylarginine--pharmacology