DNA damage activates a spatially distinct late cytoplasmic cell-cycle checkpoint network controlled by MK2-mediated RNA stabilization. [electronic resource]
- Molecular cell Oct 2010
- 34-49 p. digital
Publication Type: Journal Article; Research Support, N.I.H., Extramural
1097-4164
10.1016/j.molcel.2010.09.018 doi
3' Untranslated Regions Active Transport, Cell Nucleus Antibiotics, Antineoplastic--pharmacology Cell Cycle--drug effects Cell Cycle Proteins--genetics Cell Nucleus--enzymology Checkpoint Kinase 1 Cytoplasm--enzymology DNA Damage DNA Repair Doxorubicin--pharmacology Exoribonucleases--metabolism Feedback, Physiological HeLa Cells Head and Neck Neoplasms--enzymology Heterogeneous-Nuclear Ribonucleoproteins--metabolism Humans Intracellular Signaling Peptides and Proteins--genetics Mitosis Nuclear Proteins--genetics Phosphorylation Protein Kinases--metabolism Protein Serine-Threonine Kinases--genetics RNA Interference RNA Processing, Post-Transcriptional--drug effects RNA Stability--drug effects RNA, Messenger--metabolism RNA-Binding Proteins--metabolism Signal Transduction Time Factors Transfection Ultraviolet Rays cdc25 Phosphatases--metabolism p38 Mitogen-Activated Protein Kinases--metabolism