Siciliano, Angela <br/><br/>
Abnormal modulation of cell protective systems in response to ischemic/reperfusion injury is important in the development of mouse sickle cell hepatopathy.  [electronic resource] 

 -  Haematologica  Jan 2011 
 - 24-32 p.  digital 
<br/><br/> Publication Type: Journal Article; Research Support, Non-U.S. Gov't 
<br/><br/><label>ISSN: </label>1592-8721 
<br/><br/><label>Standard No.: </label>10.3324/haematol.2010.028506  doi 
<br/><br/><label>Subjects--Topical Terms: </label><br/>Anemia, Sickle Cell--etiology<br/>Animals<br/>Blotting, Western<br/>Cells, Cultured<br/>Cytoprotection<br/>Female<br/>HSP27 Heat-Shock Proteins--genetics<br/>HSP70 Heat-Shock Proteins--genetics<br/>Heme Oxygenase-1--genetics<br/>Hepatocytes--cytology<br/>Humans<br/>Liver Diseases--etiology<br/>Male<br/>Mice<br/>Mice, Inbred C57BL<br/>Mice, Inbred Strains<br/>Mice, Transgenic<br/>NF-kappa B--genetics<br/>Nitric Oxide--metabolism<br/>Nitric Oxide Synthase Type II--genetics<br/>Nitric Oxide Synthase Type III--genetics<br/>Peroxiredoxins--genetics<br/>RNA, Messenger--genetics<br/>Reperfusion Injury--complications<br/>Reverse Transcriptase Polymerase Chain Reaction