Jimenez-Mateos, E M <br/><br/>
Epileptic tolerance is associated with enduring neuroprotection and uncoupling of the relationship between CA3 damage, neuropeptide Y rearrangement and spontaneous seizures following intra-amygdala kainic acid-induced status epilepticus in mice.  [electronic resource] 

 -  Neuroscience  Dec 2010 
 - 556-65 p.  digital 
<br/><br/> Publication Type: Journal Article; Research Support, Non-U.S. Gov't 
<br/><br/><label>ISSN: </label>1873-7544 
<br/><br/><label>Standard No.: </label>10.1016/j.neuroscience.2010.09.003  doi 
<br/><br/><label>Subjects--Topical Terms: </label><br/>Amygdala<br/>Animals<br/>CA3 Region, Hippocampal--pathology<br/>Cell Count<br/>Cell Death<br/>Cytoprotection<br/>Kainic Acid<br/>Male<br/>Mice<br/>Mice, Inbred C57BL<br/>Mossy Fibers, Hippocampal--pathology<br/>Neurons--pathology<br/>Neuropeptide Y--metabolism<br/>Seizures--pathology<br/>Status Epilepticus--chemically induced<br/>Synapses--pathology<br/>Time Factors