Riquelme, Denise <br/><br/>
High-frequency field stimulation of primary neurons enhances ryanodine receptor-mediated Ca2+ release and generates hydrogen peroxide, which jointly stimulate NF-κB activity.  [electronic resource] 

 -  Antioxidants & redox signaling  Apr 2011 
 - 1245-59 p.  digital 
<br/><br/> Publication Type: Journal Article; Research Support, Non-U.S. Gov't 
<br/><br/><label>ISSN: </label>1557-7716 
<br/><br/><label>Standard No.: </label>10.1089/ars.2010.3238  doi 
<br/><br/><label>Subjects--Topical Terms: </label><br/>Acetylcysteine--pharmacology<br/>Animals<br/>Calcium--metabolism<br/>Cell Culture Techniques<br/>Electric Stimulation<br/>Genes, Reporter<br/>Hippocampus--cytology<br/>Hydrogen Peroxide--metabolism<br/>Luciferases, Renilla--biosynthesis<br/>NADPH Oxidases--metabolism<br/>NF-kappa B--genetics<br/>Neurons--metabolism<br/>Nitric Oxide Synthase--antagonists & inhibitors<br/>Onium Compounds--pharmacology<br/>Protein Transport<br/>Proto-Oncogene Proteins c-fos--genetics<br/>Rats<br/>Reactive Oxygen Species--metabolism<br/>Ryanodine Receptor Calcium Release Channel--metabolism<br/>Transcription Factor RelA--metabolism<br/>Transcription, Genetic<br/>Up-Regulation