Yu, Li-na <br/><br/>
Sevoflurane postconditioning reduces myocardial reperfusion injury in rat isolated hearts via activation of PI3K/Akt signaling and modulation of Bcl-2 family proteins.  [electronic resource] 

 -  Journal of Zhejiang University. Science. B  Sep 2010 
 - 661-72 p.  digital 
<br/><br/> Publication Type: Journal Article; Research Support, Non-U.S. Gov't 
<br/><br/><label>ISSN: </label>1862-1783 
<br/><br/><label>Standard No.: </label>10.1631/jzus.B1000155  doi 
<br/><br/><label>Subjects--Topical Terms: </label><br/>Androstadienes--pharmacology<br/>Anesthetics, Inhalation--antagonists & inhibitors<br/>Animals<br/>Apoptosis--drug effects<br/>Blood Pressure--physiology<br/>Blotting, Western<br/>Creatine Kinase--analysis<br/>Heart Rate--physiology<br/>Immunosuppressive Agents--pharmacology<br/>In Vitro Techniques<br/>Ischemic Postconditioning--methods<br/>L-Lactate Dehydrogenase--analysis<br/>Male<br/>Methyl Ethers--antagonists & inhibitors<br/>Myocardial Reperfusion Injury--drug therapy<br/>Proto-Oncogene Proteins c-akt--metabolism<br/>Proto-Oncogene Proteins c-bcl-2--metabolism<br/>Random Allocation<br/>Rats<br/>Rats, Sprague-Dawley<br/>Sevoflurane<br/>Signal Transduction--physiology<br/>Ventricular Pressure--physiology<br/>Wortmannin