Poulogiannis, George <br/><br/>
PARK2 deletions occur frequently in sporadic colorectal cancer and accelerate adenoma development in Apc mutant mice.  [electronic resource] 

 -  Proceedings of the National Academy of Sciences of the United States of America  Aug 2010 
 - 15145-50 p.  digital 
<br/><br/> Publication Type: Journal Article; Research Support, N.I.H., Extramural; Research Support, Non-U.S. Gov't 
<br/><br/><label>ISSN: </label>1091-6490 
<br/><br/><label>Standard No.: </label>10.1073/pnas.1009941107  doi 
<br/><br/><label>Subjects--Topical Terms: </label><br/>Adenomatous Polyposis Coli--etiology<br/>Animals<br/>Base Sequence<br/>Cell Line, Tumor<br/>Cell Proliferation<br/>Chromosomes, Human, Pair 6--genetics<br/>Cocarcinogenesis<br/>Colorectal Neoplasms--genetics<br/>DNA Methylation<br/>DNA Primers--genetics<br/>DNA, Neoplasm--chemistry<br/>Gene Deletion<br/>Gene Dosage<br/>Genes, APC<br/>Genes, Tumor Suppressor<br/>Heterozygote<br/>Humans<br/>Mice<br/>Mice, Inbred C57BL<br/>Mice, Knockout<br/>Mice, Mutant Strains<br/>Models, Molecular<br/>Mutation<br/>Spectral Karyotyping<br/>Ubiquitin-Protein Ligases--chemistry