Reiniger, Nina <br/><br/>
Deletion of the receptor for advanced glycation end products reduces glomerulosclerosis and preserves renal function in the diabetic OVE26 mouse.  [electronic resource] 

 -  Diabetes  Aug 2010 
 - 2043-54 p.  digital 
<br/><br/> Publication Type: Journal Article 
<br/><br/><label>ISSN: </label>1939-327X 
<br/><br/><label>Standard No.: </label>10.2337/db09-1766  doi 
<br/><br/><label>Subjects--Topical Terms: </label><br/>Animals<br/>Blood Glucose--metabolism<br/>Chickens<br/>Diabetic Nephropathies--genetics<br/>Disease Models, Animal<br/>Gene Deletion<br/>Glomerular Mesangium--metabolism<br/>Glycated Hemoglobin--metabolism<br/>Kidney--metabolism<br/>Mice<br/>Mice, Inbred Strains<br/>Pyruvaldehyde--metabolism<br/>Receptor for Advanced Glycation End Products<br/>Receptors, Immunologic--deficiency<br/>Renal Insufficiency--genetics