Takeuchi, Koji <br/><br/>
Endogenous prostaglandin E2 accelerates healing of indomethacin-induced small intestinal lesions through upregulation of vascular endothelial growth factor expression by activation of EP4 receptors.  [electronic resource] 

 -  Journal of gastroenterology and hepatology  May 2010 
 - S67-74 p.  digital 
<br/><br/> Publication Type: Journal Article; Research Support, Non-U.S. Gov't 
<br/><br/><label>ISSN: </label>1440-1746 
<br/><br/><label>Standard No.: </label>10.1111/j.1440-1746.2010.06222.x  doi 
<br/><br/><label>Subjects--Topical Terms: </label><br/>16,16-Dimethylprostaglandin E2--pharmacology<br/>Animals<br/>Cyclooxygenase 1--genetics<br/>Cyclooxygenase 2--genetics<br/>Dinoprostone--metabolism<br/>Disease Models, Animal<br/>Gene Expression Regulation, Enzymologic<br/>Indomethacin<br/>Intestinal Mucosa--drug effects<br/>Intestine, Small--drug effects<br/>Male<br/>Membrane Proteins--genetics<br/>Naphthalenes--pharmacology<br/>Neovascularization, Physiologic--drug effects<br/>Peptic Ulcer--chemically induced<br/>Phenylbutyrates--pharmacology<br/>RNA, Messenger--metabolism<br/>Rats<br/>Rats, Sprague-Dawley<br/>Receptors, Prostaglandin E--agonists<br/>Receptors, Prostaglandin E, EP4 Subtype<br/>Severity of Illness Index<br/>Time Factors<br/>Up-Regulation<br/>Vascular Endothelial Growth Factor A--metabolism<br/>Wound Healing--drug effects