ROS accumulation and IGF-IR inhibition contribute to fenofibrate/PPARalpha -mediated inhibition of glioma cell motility in vitro. [electronic resource]
- Molecular cancer Jun 2010
- 159 p. digital
Publication Type: Journal Article; Research Support, N.I.H., Extramural; Research Support, Non-U.S. Gov't
1476-4598
10.1186/1476-4598-9-159 doi
Adenosine Triphosphate--biosynthesis Base Sequence Brain Neoplasms--metabolism Cell Line, Tumor Cell Movement--drug effects Fenofibrate--pharmacology Glioma--metabolism Humans PPAR alpha--physiology RNA, Small Interfering Reactive Oxygen Species--metabolism Receptor, IGF Type 1--antagonists & inhibitors