Luccini, Elisa <br/><br/>
Ionic dysregulations typical of ischemia provoke release of glycine and GABA by multiple mechanisms.  [electronic resource] 

 -  Journal of neurochemistry  Aug 2010 
 - 1074-84 p.  digital 
<br/><br/> Publication Type: Journal Article; Research Support, Non-U.S. Gov't 
<br/><br/><label>ISSN: </label>1471-4159 
<br/><br/><label>Standard No.: </label>10.1111/j.1471-4159.2010.06829.x  doi 
<br/><br/><label>Subjects--Topical Terms: </label><br/>Animals<br/>Brain Ischemia--metabolism<br/>Exocytosis--drug effects<br/>Glycine--metabolism<br/>Hippocampus--drug effects<br/>Mice<br/>Mitochondria--metabolism<br/>Neurotransmitter Transport Proteins--metabolism<br/>Potassium Chloride--pharmacology<br/>Presynaptic Terminals--drug effects<br/>Sodium-Calcium Exchanger--drug effects<br/>Spinal Cord--drug effects<br/>Subcellular Fractions--drug effects<br/>Synaptosomes--drug effects<br/>Veratridine--pharmacology<br/>gamma-Aminobutyric Acid--metabolism