Kong, Hong-liang <br/><br/>
Ginsenoside Rb1 protects cardiomyocytes against CoCl2-induced apoptosis in neonatal rats by inhibiting mitochondria permeability transition pore opening.  [electronic resource] 

 -  Acta pharmacologica Sinica  Jun 2010 
 - 687-95 p.  digital 
<br/><br/> Publication Type: Journal Article 
<br/><br/><label>ISSN: </label>1745-7254 
<br/><br/><label>Standard No.: </label>10.1038/aps.2010.52  doi 
<br/><br/><label>Subjects--Topical Terms: </label><br/>Androstadienes--pharmacology<br/>Animals<br/>Animals, Newborn<br/>Apoptosis--drug effects<br/>Blotting, Western<br/>Cardiotonic Agents--pharmacology<br/>Caspase 3--analysis<br/>Cell Hypoxia--drug effects<br/>Cells, Cultured<br/>Cobalt--toxicity<br/>Cytochromes c--analysis<br/>Flow Cytometry<br/>Ginsenosides--pharmacology<br/>Glycogen Synthase Kinase 3--metabolism<br/>Glycogen Synthase Kinase 3 beta<br/>Immunohistochemistry<br/>Mitochondria, Heart--drug effects<br/>Mitochondrial Membrane Transport Proteins--metabolism<br/>Mitochondrial Permeability Transition Pore<br/>Myocytes, Cardiac--cytology<br/>Protein Kinase Inhibitors--pharmacology<br/>Random Allocation<br/>Rats<br/>Wortmannin