Loizzo, Stefano <br/><br/>
Sexual dimorphic evolution of metabolic programming in non-genetic non-alimentary mild metabolic syndrome model in mice depends on feed-back mechanisms integrity for pro-opiomelanocortin-derived endogenous substances.  [electronic resource] 

 -  Peptides  Aug 2010 
 - 1598-605 p.  digital 
<br/><br/> Publication Type: Journal Article 
<br/><br/><label>ISSN: </label>1873-5169 
<br/><br/><label>Standard No.: </label>10.1016/j.peptides.2010.05.006  doi 
<br/><br/><label>Subjects--Topical Terms: </label><br/>11-beta-Hydroxysteroid Dehydrogenase Type 1--genetics<br/>11-beta-Hydroxysteroid Dehydrogenase Type 2--genetics<br/>Adrenocorticotropic Hormone--blood<br/>Animals<br/>Animals, Newborn<br/>Brain--metabolism<br/>Corticosterone--blood<br/>Diabetes Mellitus, Type 2--blood<br/>Feedback, Physiological<br/>Female<br/>Gene Expression Regulation, Enzymologic<br/>Handling, Psychological<br/>Hypothalamo-Hypophyseal System--metabolism<br/>Lipid Metabolism--physiology<br/>Male<br/>Metabolic Syndrome--blood<br/>Mice<br/>Pain Threshold--physiology<br/>Pituitary-Adrenal System--metabolism<br/>Pro-Opiomelanocortin--metabolism<br/>RNA, Messenger--metabolism<br/>Random Allocation<br/>Sex Characteristics<br/>Stress, Psychological--complications