Moriel-Carretero, Marķa <br/><br/>
A postincision-deficient TFIIH causes replication fork breakage and uncovers alternative Rad51- or Pol32-mediated restart mechanisms.  [electronic resource] 

 -  Molecular cell  Mar 2010 
 - 690-701 p.  digital 
<br/><br/> Publication Type: Journal Article; Research Support, Non-U.S. Gov't 
<br/><br/><label>ISSN: </label>1097-4164 
<br/><br/><label>Standard No.: </label>10.1016/j.molcel.2010.02.008  doi 
<br/><br/><label>Subjects--Topical Terms: </label><br/>DNA Breaks, Double-Stranded<br/>DNA Helicases--genetics<br/>DNA Repair--radiation effects<br/>DNA Repair Enzymes--genetics<br/>DNA Replication--radiation effects<br/>DNA, Fungal--biosynthesis<br/>DNA-Binding Proteins--genetics<br/>DNA-Directed DNA Polymerase--genetics<br/>Dose-Response Relationship, Radiation<br/>Endodeoxyribonucleases--genetics<br/>Endonucleases--genetics<br/>Genomic Instability<br/>Genotype<br/>Humans<br/>Mutation<br/>Phenotype<br/>Rad51 Recombinase--genetics<br/>Rad52 DNA Repair and Recombination Protein--genetics<br/>Radiation Tolerance<br/>Recombination, Genetic--radiation effects<br/>Replication Protein C--genetics<br/>Saccharomyces cerevisiae--genetics<br/>Saccharomyces cerevisiae Proteins--genetics<br/>Time Factors<br/>Transcription Factor TFIIH--genetics<br/>Ultraviolet Rays