Mimura, Kazuya <br/><br/>
Nicotine restores endothelial dysfunction caused by excess sFlt1 and sEng in an in vitro model of preeclamptic vascular endothelium: a possible therapeutic role of nicotinic acetylcholine receptor (nAChR) agonists for preeclampsia.  [electronic resource] 

 -  American journal of obstetrics and gynecology  May 2010 
 - 464.e1-6 p.  digital 
<br/><br/> Publication Type: Journal Article; Research Support, Non-U.S. Gov't 
<br/><br/><label>ISSN: </label>1097-6868 
<br/><br/><label>Standard No.: </label>10.1016/j.ajog.2010.01.037  doi 
<br/><br/><label>Subjects--Topical Terms: </label><br/>Antigens, CD--physiology<br/>Endoglin<br/>Endothelium, Vascular--drug effects<br/>Enzyme-Linked Immunosorbent Assay<br/>Female<br/>Humans<br/>Nicotine--pharmacology<br/>Nicotinic Agonists--pharmacology<br/>Pre-Eclampsia--drug therapy<br/>Pregnancy<br/>Receptors, Cell Surface--physiology<br/>Receptors, Cholinergic--drug effects<br/>Receptors, Nicotinic--drug effects<br/>Vascular Endothelial Growth Factor Receptor-1--physiology<br/>Wound Healing--physiology