Watanabe, Yusuke <br/><br/>
IL-1beta suppresses the formation of osteoclasts by increasing OPG production via an autocrine mechanism involving celecoxib-related prostaglandins in chondrocytes.  [electronic resource] 

 -  Mediators of inflammation  2009 
 - 308596 p.  digital 
<br/><br/> Publication Type: Journal Article; Research Support, Non-U.S. Gov't 
<br/><br/><label>ISSN: </label>1466-1861 
<br/><br/><label>Standard No.: </label>10.1155/2009/308596  doi 
<br/><br/><label>Subjects--Topical Terms: </label><br/>Animals<br/>Autocrine Communication--drug effects<br/>Base Sequence<br/>Celecoxib<br/>Cell Differentiation--drug effects<br/>Cell Line<br/>Cells, Cultured<br/>Chondrocytes--cytology<br/>Culture Media, Conditioned<br/>DNA Primers--genetics<br/>Dinoprostone--metabolism<br/>Gene Expression--drug effects<br/>Humans<br/>Inflammation Mediators--metabolism<br/>Interleukin-1beta--pharmacology<br/>Macrophage Colony-Stimulating Factor--biosynthesis<br/>Mice<br/>Osteoclasts--cytology<br/>Osteoprotegerin--biosynthesis<br/>Prostaglandins--metabolism<br/>Pyrazoles--pharmacology<br/>RANK Ligand--biosynthesis<br/>Sulfonamides--pharmacology