Samuel, Samson Mathews <br/><br/>
Thioredoxin-1 gene therapy enhances angiogenic signaling and reduces ventricular remodeling in infarcted myocardium of diabetic rats.  [electronic resource] 

 -  Circulation  Mar 2010 
 - 1244-55 p.  digital 
<br/><br/> Publication Type: Journal Article; Research Support, N.I.H., Extramural 
<br/><br/><label>ISSN: </label>1524-4539 
<br/><br/><label>Standard No.: </label>10.1161/CIRCULATIONAHA.109.872481  doi 
<br/><br/><label>Subjects--Topical Terms: </label><br/>Animals<br/>Apoptosis<br/>Cells, Cultured<br/>Diabetes Mellitus, Experimental--complications<br/>Endothelial Cells--metabolism<br/>Fibrosis<br/>Genetic Therapy<br/>Heme Oxygenase-1--metabolism<br/>Humans<br/>JNK Mitogen-Activated Protein Kinases--analysis<br/>Male<br/>Myocardial Infarction--pathology<br/>Myocardium--metabolism<br/>Neovascularization, Physiologic<br/>Oxidative Stress<br/>Rats<br/>Rats, Sprague-Dawley<br/>Signal Transduction<br/>Streptozocin<br/>Thioredoxins--analysis<br/>Transfection<br/>Vascular Endothelial Growth Factor A--analysis<br/>Ventricular Remodeling<br/>p38 Mitogen-Activated Protein Kinases--analysis