Ii, Masaaki <br/><br/>
Notch signaling regulates endothelial progenitor cell activity during recovery from arterial injury in hypercholesterolemic mice.  [electronic resource] 

 -  Circulation  Mar 2010 
 - 1104-12 p.  digital 
<br/><br/> Publication Type: Journal Article; Research Support, N.I.H., Extramural 
<br/><br/><label>ISSN: </label>1524-4539 
<br/><br/><label>Standard No.: </label>10.1161/CIRCULATIONAHA.105.553917  doi 
<br/><br/><label>Subjects--Topical Terms: </label><br/>Animals<br/>Apolipoproteins E--deficiency<br/>Atherosclerosis--etiology<br/>Bone Marrow Transplantation<br/>Carotid Artery Injuries--complications<br/>Cell Movement<br/>Cholesterol--blood<br/>Endothelial Cells--pathology<br/>Gene Expression Regulation<br/>Genotype<br/>Hypercholesterolemia--complications<br/>Male<br/>Mesenchymal Stem Cells--pathology<br/>Mice<br/>Mice, Inbred C57BL<br/>Mice, Knockout<br/>Nitric Oxide Synthase Type III--biosynthesis<br/>RNA, Messenger--biosynthesis<br/>Radiation Chimera<br/>Receptor, Notch1--biosynthesis<br/>Signal Transduction<br/>Vascular Endothelial Growth Factor A--biosynthesis