Jin, Yi <br/><br/>
Mice deficient in Mkp-1 develop more severe pulmonary hypertension and greater lung protein levels of arginase in response to chronic hypoxia.  [electronic resource] 

 -  American journal of physiology. Heart and circulatory physiology  May 2010 
 - H1518-28 p.  digital 
<br/><br/> Publication Type: Journal Article; Research Support, N.I.H., Extramural 
<br/><br/><label>ISSN: </label>1522-1539 
<br/><br/><label>Standard No.: </label>10.1152/ajpheart.00813.2009  doi 
<br/><br/><label>Subjects--Topical Terms: </label><br/>Animals<br/>Arginase--metabolism<br/>Blotting, Western<br/>Body Weight--physiology<br/>Chronic Disease<br/>Dual Specificity Phosphatase 1--genetics<br/>Hypertension, Pulmonary--genetics<br/>Hypoxia--genetics<br/>Lung--metabolism<br/>Mice<br/>Mice, Inbred C57BL<br/>Mice, Knockout<br/>Myocardium--metabolism<br/>Nitrates--metabolism<br/>Nitric Oxide Synthase Type I--biosynthesis<br/>Nitric Oxide Synthase Type II--biosynthesis<br/>Nitric Oxide Synthase Type III--biosynthesis<br/>Nitrites--metabolism<br/>Proliferating Cell Nuclear Antigen--metabolism<br/>Proteins--metabolism<br/>Ventricular Function, Right--physiology<br/>p38 Mitogen-Activated Protein Kinases--metabolism