Wang, Shuangxi
AMPKalpha2 deletion causes aberrant expression and activation of NAD(P)H oxidase and consequent endothelial dysfunction in vivo: role of 26S proteasomes. [electronic resource]
- Circulation research Apr 2010
- 1117-28 p. digital
Publication Type: Journal Article; Research Support, N.I.H., Extramural; Research Support, Non-U.S. Gov't
ISSN: 1524-4571
Standard No.: 10.1161/CIRCRESAHA.109.212530 doi
Subjects--Topical Terms: AMP-Activated Protein Kinases--antagonists & inhibitors Acetophenones--pharmacology Acetylcholine--pharmacology Aminoimidazole Carboxamide--analogs & derivatives Animals Antioxidants--pharmacology Cells, Cultured Cyclic N-Oxides--pharmacology Dose-Response Relationship, Drug Endothelial Cells--drug effects Endothelium, Vascular--drug effects Enzyme Activation Enzyme Inhibitors--pharmacology Gene Deletion Gene Expression Regulation, Enzymologic Humans I-kappa B Proteins--metabolism Inflammation Mediators--blood Interferon-gamma--blood Interleukin-2--blood Isoenzymes Leupeptins--pharmacology Male Mice Mice, Inbred C57BL Mice, Knockout NADPH Oxidases--antagonists & inhibitors NF-KappaB Inhibitor alpha NF-kappa B--metabolism Nitroprusside--pharmacology Oxidative Stress--drug effects Proteasome Endopeptidase Complex--metabolism Proteasome Inhibitors RNA Interference Receptors, LDL--deficiency Ribonucleotides--pharmacology Spin Labels Superoxide Dismutase--metabolism Superoxides--metabolism Transfection Vasodilation--drug effects Vasodilator Agents--pharmacology