Wang, Shuangxi

AMPKalpha2 deletion causes aberrant expression and activation of NAD(P)H oxidase and consequent endothelial dysfunction in vivo: role of 26S proteasomes. [electronic resource] - Circulation research Apr 2010 - 1117-28 p. digital

Publication Type: Journal Article; Research Support, N.I.H., Extramural; Research Support, Non-U.S. Gov't

1524-4571

10.1161/CIRCRESAHA.109.212530 doi


AMP-Activated Protein Kinases--antagonists & inhibitors
Acetophenones--pharmacology
Acetylcholine--pharmacology
Aminoimidazole Carboxamide--analogs & derivatives
Animals
Antioxidants--pharmacology
Cells, Cultured
Cyclic N-Oxides--pharmacology
Dose-Response Relationship, Drug
Endothelial Cells--drug effects
Endothelium, Vascular--drug effects
Enzyme Activation
Enzyme Inhibitors--pharmacology
Gene Deletion
Gene Expression Regulation, Enzymologic
Humans
I-kappa B Proteins--metabolism
Inflammation Mediators--blood
Interferon-gamma--blood
Interleukin-2--blood
Isoenzymes
Leupeptins--pharmacology
Male
Mice
Mice, Inbred C57BL
Mice, Knockout
NADPH Oxidases--antagonists & inhibitors
NF-KappaB Inhibitor alpha
NF-kappa B--metabolism
Nitroprusside--pharmacology
Oxidative Stress--drug effects
Proteasome Endopeptidase Complex--metabolism
Proteasome Inhibitors
RNA Interference
Receptors, LDL--deficiency
Ribonucleotides--pharmacology
Spin Labels
Superoxide Dismutase--metabolism
Superoxides--metabolism
Transfection
Vasodilation--drug effects
Vasodilator Agents--pharmacology