Naiki-Ito, Aya <br/><br/>
Gap junction dysfunction reduces acetaminophen hepatotoxicity with impact on apoptotic signaling and connexin 43 protein induction in rat.  [electronic resource] 

 -  Toxicologic pathology  Feb 2010 
 - 280-6 p.  digital 
<br/><br/> Publication Type: Journal Article 
<br/><br/><label>ISSN: </label>1533-1601 
<br/><br/><label>Standard No.: </label>10.1177/0192623309357951  doi 
<br/><br/><label>Subjects--Topical Terms: </label><br/>Acetaminophen--toxicity<br/>Analgesics, Non-Narcotic--toxicity<br/>Animals<br/>Apoptosis--drug effects<br/>Chemical and Drug Induced Liver Injury--metabolism<br/>Connexin 43--drug effects<br/>Connexins--metabolism<br/>Female<br/>Gap Junctions--drug effects<br/>Male<br/>Rats<br/>Rats, Sprague-Dawley<br/>Signal Transduction--drug effects<br/>Gap Junction beta-1 Protein