Smith, Patrice D <br/><br/>
SOCS3 deletion promotes optic nerve regeneration in vivo.  [electronic resource] 

 -  Neuron  Dec 2009 
 - 617-23 p.  digital 
<br/><br/> Publication Type: Journal Article; Research Support, Non-U.S. Gov't 
<br/><br/><label>ISSN: </label>1097-4199 
<br/><br/><label>Standard No.: </label>10.1016/j.neuron.2009.11.021  doi 
<br/><br/><label>Subjects--Topical Terms: </label><br/>Analysis of Variance<br/>Animals<br/>Animals, Newborn<br/>Axons--drug effects<br/>Carrier Proteins--metabolism<br/>Cholera Toxin--metabolism<br/>Ciliary Neurotrophic Factor--genetics<br/>Cytokine Receptor gp130--deficiency<br/>Disease Models, Animal<br/>Dose-Response Relationship, Drug<br/>Gene Expression Regulation--genetics<br/>Green Fluorescent Proteins--genetics<br/>Injections, Intraventricular--methods<br/>Mice<br/>Mice, Inbred C57BL<br/>Mice, Transgenic<br/>Nerve Regeneration--drug effects<br/>Optic Nerve Injuries--drug therapy<br/>Organ Culture Techniques<br/>Phosphotransferases (Alcohol Group Acceptor)--metabolism<br/>Retinal Ganglion Cells--pathology<br/>Suppressor of Cytokine Signaling 3 Protein<br/>Suppressor of Cytokine Signaling Proteins--deficiency<br/>TOR Serine-Threonine Kinases<br/>Time Factors<br/>Tubulin--metabolism