Chen, Ku-Chung <br/><br/>
Taiwan cobra phospholipase A2-elicited JNK activation is responsible for autocrine fas-mediated cell death and modulating Bcl-2 and Bax protein expression in human leukemia K562 cells.  [electronic resource] 

 -  Journal of cellular biochemistry  Jan 2010 
 - 245-54 p.  digital 
<br/><br/> Publication Type: Journal Article; Research Support, Non-U.S. Gov't 
<br/><br/><label>ISSN: </label>1097-4644 
<br/><br/><label>Standard No.: </label>10.1002/jcb.22404  doi 
<br/><br/><label>Subjects--Topical Terms: </label><br/>Apoptosis--physiology<br/>Blotting, Western<br/>Cell Separation<br/>Cell Survival--drug effects<br/>Elapid Venoms--enzymology<br/>Enzyme Activation--physiology<br/>Enzyme Inhibitors--pharmacology<br/>Fas Ligand Protein--metabolism<br/>Flow Cytometry<br/>Humans<br/>K562 Cells<br/>Leukemia--metabolism<br/>MAP Kinase Kinase 4--metabolism<br/>Membrane Potential, Mitochondrial--drug effects<br/>Phospholipases A2--metabolism<br/>Proto-Oncogene Proteins c-bcl-2--biosynthesis<br/>RNA Interference<br/>Signal Transduction--drug effects<br/>Transfection<br/>bcl-2-Associated X Protein--biosynthesis<br/>fas Receptor--metabolism