Xu, Chuan-Rui <br/><br/>
Bmi1 functions as an oncogene independent of Ink4A/Arf repression in hepatic carcinogenesis.  [electronic resource] 

 -  Molecular cancer research : MCR  Dec 2009 
 - 1937-45 p.  digital 
<br/><br/> Publication Type: Journal Article; Research Support, N.I.H., Extramural 
<br/><br/><label>ISSN: </label>1557-3125 
<br/><br/><label>Standard No.: </label>10.1158/1541-7786.MCR-09-0333  doi 
<br/><br/><label>Subjects--Topical Terms: </label><br/>Animals<br/>Carcinoma, Hepatocellular--genetics<br/>Cell Cycle<br/>Cell Line, Tumor<br/>Cell Proliferation<br/>Cyclin-Dependent Kinase Inhibitor p16--genetics<br/>Down-Regulation--genetics<br/>Enzyme Activation<br/>Gene Expression Regulation, Neoplastic<br/>Gene Knockdown Techniques<br/>Hepatocytes--metabolism<br/>Humans<br/>Liver Neoplasms--genetics<br/>Mice<br/>Nuclear Proteins--genetics<br/>Polycomb Repressive Complex 1<br/>Proto-Oncogene Mas<br/>Proto-Oncogene Proteins--genetics<br/>RNA, Small Interfering--metabolism<br/>Repressor Proteins--genetics<br/>Tumor Suppressor Protein p14ARF--genetics<br/>Up-Regulation--genetics<br/>ras Proteins--metabolism