Roussel, Lucie <br/><br/>
IL-17 primes airway epithelial cells lacking functional Cystic Fibrosis Transmembrane conductance Regulator (CFTR) to increase NOD1 responses.  [electronic resource] 

 -  Biochemical and biophysical research communications  Jan 2010 
 - 505-9 p.  digital 
<br/><br/> Publication Type: Journal Article; Research Support, Non-U.S. Gov't 
<br/><br/><label>ISSN: </label>1090-2104 
<br/><br/><label>Standard No.: </label>10.1016/j.bbrc.2009.11.088  doi 
<br/><br/><label>Subjects--Topical Terms: </label><br/>Adolescent<br/>Cells, Cultured<br/>Cystic Fibrosis--complications<br/>Cystic Fibrosis Transmembrane Conductance Regulator--genetics<br/>Epithelial Cells--immunology<br/>Humans<br/>Interleukin-17--metabolism<br/>Interleukin-8--metabolism<br/>Lung--immunology<br/>Neutrophils--immunology<br/>Nod1 Signaling Adaptor Protein--biosynthesis<br/>Pseudomonas Infections--immunology<br/>Pseudomonas aeruginosa<br/>Receptors, Interleukin-17--biosynthesis<br/>Respiratory Mucosa--immunology<br/>Toll-Like Receptor 4--biosynthesis