Sustained deficiency of mitochondrial complex I activity during long periods of survival after seizures induced in immature rats by homocysteic acid. [electronic resource]
- Neurochemistry international Feb 2010
- 394-403 p. digital
Publication Type: Journal Article; Research Support, Non-U.S. Gov't
1872-9754
10.1016/j.neuint.2009.11.011 doi
Aldehydes--metabolism Animals Animals, Newborn Cerebral Cortex--metabolism Convulsants--toxicity Disease Models, Animal Down-Regulation--drug effects Electron Transport Complex I--drug effects Energy Metabolism--drug effects Epilepsy--metabolism Excitatory Amino Acid Agonists--pharmacology Free Radical Scavengers--pharmacology Homocysteine--analogs & derivatives Male Metabolic Networks and Pathways--physiology Mitochondria--drug effects Mitochondrial Diseases--chemically induced Oxidative Stress--drug effects Rats Rats, Wistar Seizures--chemically induced Survival Rate Time Factors Tyrosine--analogs & derivatives