Teoh, Narci
Induction of p53 renders ATM-deficient mice refractory to hepatocarcinogenesis. [electronic resource]
- Gastroenterology Mar 2010
- 1155-65.e1-2 p. digital
Publication Type: Journal Article; Research Support, N.I.H., Extramural; Research Support, Non-U.S. Gov't
ISSN: 1528-0012
Standard No.: 10.1053/j.gastro.2009.11.008 doi
Subjects--Topical Terms: Animals Apoptosis Ataxia Telangiectasia Mutated Proteins Carcinoma, Hepatocellular--chemically induced Caspase 3--metabolism Cell Cycle Cell Cycle Proteins--genetics Cell Transformation, Neoplastic--chemically induced Cellular Senescence Checkpoint Kinase 1 Chromosome Aberrations Cyclin-Dependent Kinase Inhibitor p16--metabolism DNA-Binding Proteins--deficiency Diethylnitrosamine Disease Models, Animal Gene Expression Regulation, Neoplastic Heterozygote Karyotyping Liver Neoplasms--chemically induced Male Mice Mice, Knockout Protein Kinases--metabolism Protein Serine-Threonine Kinases--deficiency Signal Transduction Telomerase--metabolism Time Factors Tumor Suppressor Protein p53--genetics Tumor Suppressor Proteins--deficiency Up-Regulation