l'Hoste, Sebastien <br/><br/>
CFTR mediates apoptotic volume decrease and cell death by controlling glutathione efflux and ROS production in cultured mice proximal tubules.  [electronic resource] 

 -  American journal of physiology. Renal physiology  Feb 2010 
 - F435-53 p.  digital 
<br/><br/> Publication Type: Journal Article 
<br/><br/><label>ISSN: </label>1522-1466 
<br/><br/><label>Standard No.: </label>10.1152/ajprenal.00286.2009  doi 
<br/><br/><label>Subjects--Topical Terms: </label><br/>Animals<br/>Antibodies, Monoclonal<br/>Apoptosis--drug effects<br/>Caspase 3--metabolism<br/>Cell Death<br/>Cell Line, Transformed<br/>Chloride Channels--metabolism<br/>Cystic Fibrosis Transmembrane Conductance Regulator--deficiency<br/>DNA, Complementary<br/>Down-Regulation<br/>Electric Conductivity<br/>Enzyme Activation--drug effects<br/>Glutathione--metabolism<br/>Glutathione Disulfide--metabolism<br/>Humans<br/>Kidney Tubules, Proximal--cytology<br/>Mice<br/>Mice, Knockout<br/>Reactive Oxygen Species--metabolism<br/>Staurosporine--pharmacology<br/>Transfection<br/>Tumor Necrosis Factor-alpha--pharmacology