Fujimura, Masatake <br/><br/>
Methylmercury induces neuropathological changes with tau hyperphosphorylation mainly through the activation of the c-jun-N-terminal kinase pathway in the cerebral cortex, but not in the hippocampus of the mouse brain.  [electronic resource] 

 -  Neurotoxicology  Nov 2009 
 - 1000-7 p.  digital 
<br/><br/> Publication Type: Journal Article; Research Support, Non-U.S. Gov't 
<br/><br/><label>ISSN: </label>1872-9711 
<br/><br/><label>Standard No.: </label>10.1016/j.neuro.2009.08.001  doi 
<br/><br/><label>Subjects--Topical Terms: </label><br/>Animals<br/>Cerebral Cortex--drug effects<br/>Disease Models, Animal<br/>Hippocampus--drug effects<br/>JNK Mitogen-Activated Protein Kinases--metabolism<br/>Male<br/>Mercury Poisoning, Nervous System--enzymology<br/>Methylmercury Compounds--blood<br/>Mice<br/>Mice, Inbred C57BL<br/>Signal Transduction--drug effects<br/>Time Factors<br/>tau Proteins--metabolism