Jin, H K <br/><br/>
Amyloid beta-derived neuroplasticity in bone marrow-derived mesenchymal stem cells is mediated by NPY and 5-HT2B receptors via ERK1/2 signalling pathways.  [electronic resource] 

 -  Cell proliferation  Oct 2009 
 - 571-86 p.  digital 
<br/><br/> Publication Type: Journal Article; Research Support, Non-U.S. Gov't 
<br/><br/><label>ISSN: </label>1365-2184 
<br/><br/><label>Standard No.: </label>10.1111/j.1365-2184.2009.00625.x  doi 
<br/><br/><label>Subjects--Topical Terms: </label><br/>Alzheimer Disease--metabolism<br/>Amyloid beta-Peptides--metabolism<br/>Animals<br/>Apoptosis--drug effects<br/>Bone Marrow Cells--cytology<br/>Cell Differentiation--drug effects<br/>Cell Division--drug effects<br/>Cell Survival--drug effects<br/>Cells, Cultured<br/>Dynamins--metabolism<br/>MAP Kinase Signaling System--physiology<br/>Mesenchymal Stem Cells--cytology<br/>Mice<br/>Mice, Inbred C57BL<br/>Mice, Mutant Strains<br/>Mitogen-Activated Protein Kinase 1--metabolism<br/>Mitogen-Activated Protein Kinase 3--metabolism<br/>Neuronal Plasticity--physiology<br/>Neuropeptide Y--genetics<br/>Peptide Fragments--metabolism<br/>Phosphatidylinositol 3-Kinases--metabolism<br/>Receptor, Serotonin, 5-HT2B--metabolism<br/>Receptors, Neuropeptide Y--metabolism