Gourronc, Francoise A <br/><br/>
Proliferative defects in dyskeratosis congenita skin keratinocytes are corrected by expression of the telomerase reverse transcriptase, TERT, or by activation of endogenous telomerase through expression of papillomavirus E6/E7 or the telomerase RNA component, TERC.  [electronic resource] 

 -  Experimental dermatology  Mar 2010 
 - 279-88 p.  digital 
<br/><br/> Publication Type: Journal Article; Research Support, N.I.H., Extramural; Research Support, Non-U.S. Gov't 
<br/><br/><label>ISSN: </label>1600-0625 
<br/><br/><label>Standard No.: </label>10.1111/j.1600-0625.2009.00916.x  doi 
<br/><br/><label>Subjects--Topical Terms: </label><br/>Adult<br/>Base Sequence<br/>Cell Proliferation<br/>Colony-Forming Units Assay<br/>DNA Primers--genetics<br/>Dyskeratosis Congenita--enzymology<br/>Enzyme Activation<br/>Female<br/>Fibroblasts--enzymology<br/>Gene Expression<br/>Humans<br/>In Vitro Techniques<br/>Keratinocytes--enzymology<br/>Male<br/>Mutation<br/>Oncogene Proteins, Viral--genetics<br/>Papillomavirus E7 Proteins--genetics<br/>RNA--genetics<br/>Repressor Proteins--genetics<br/>Telomerase--genetics<br/>Telomere--genetics<br/>Transduction, Genetic<br/>Up-Regulation<br/>Young Adult