Lutz, Sarah E <br/><br/>
Deletion of astrocyte connexins 43 and 30 leads to a dysmyelinating phenotype and hippocampal CA1 vacuolation.  [electronic resource] 

 -  The Journal of neuroscience : the official journal of the Society for Neuroscience  Jun 2009 
 - 7743-52 p.  digital 
<br/><br/> Publication Type: Journal Article; Research Support, N.I.H., Extramural; Research Support, Non-U.S. Gov't 
<br/><br/><label>ISSN: </label>1529-2401 
<br/><br/><label>Standard No.: </label>10.1523/JNEUROSCI.0341-09.2009  doi 
<br/><br/><label>Subjects--Topical Terms: </label><br/>Animals<br/>Animals, Newborn<br/>Astrocytes--metabolism<br/>Basic Helix-Loop-Helix Transcription Factors--metabolism<br/>Calcium-Binding Proteins<br/>Cell Proliferation<br/>Connexin 30<br/>Connexin 43--deficiency<br/>Connexins--deficiency<br/>DNA-Binding Proteins--metabolism<br/>Demyelinating Diseases--complications<br/>Disease Models, Animal<br/>Gap Junctions--pathology<br/>Glial Fibrillary Acidic Protein--genetics<br/>Hippocampus--pathology<br/>In Situ Nick-End Labeling--methods<br/>Memory Disorders--genetics<br/>Mice<br/>Mice, Inbred C57BL<br/>Mice, Knockout<br/>Microfilament Proteins<br/>Microscopy, Electron--methods<br/>Myelin Basic Protein--metabolism<br/>Myelin Sheath--pathology<br/>Nerve Tissue Proteins--metabolism<br/>Oligodendrocyte Transcription Factor 2<br/>Phenotype