Natarajan, Ramesh
Prolyl hydroxylase inhibition attenuates post-ischemic cardiac injury via induction of endoplasmic reticulum stress genes. [electronic resource]
- Vascular pharmacology
- 110-8 p. digital
Publication Type: Journal Article; Research Support, N.I.H., Extramural
1879-3649
10.1016/j.vph.2009.05.007 doi
Activating Transcription Factor 4--genetics
Amino Acids, Dicarboxylic--pharmacology
Analysis of Variance
Animals
Apoptosis--drug effects
Biomarkers--metabolism
Caspase 3--metabolism
Cell Hypoxia--drug effects
Cell Line
DNA-Binding Proteins--genetics
Endoplasmic Reticulum--drug effects
Endoplasmic Reticulum Chaperone BiP
Endothelial Cells--drug effects
Enzyme Activation
Enzyme Inhibitors--pharmacology
Gene Expression Regulation--drug effects
Gene Silencing
Heat-Shock Proteins--genetics
Heme Oxygenase-1--genetics
Male
Mice
Microscopy, Fluorescence
Microvessels
Myocardial Reperfusion Injury--drug therapy
Myocardium--metabolism
Myocytes, Cardiac--drug effects
NF-E2-Related Factor 2--immunology
Phosphorylation
Procollagen-Proline Dioxygenase--antagonists & inhibitors
Protein Transport
RNA, Messenger--metabolism
RNA, Small Interfering
Regulatory Factor X Transcription Factors
Reverse Transcriptase Polymerase Chain Reaction
Stress, Physiological--drug effects
Transcription Factor CHOP--genetics
Transcription Factors--genetics
eIF-2 Kinase--metabolism