Sathishkumar, K <br/><br/>
Cholesterol secoaldehyde induces apoptosis in H9c2 cardiomyoblasts through reactive oxygen species involving mitochondrial and death receptor pathways.  [electronic resource] 

 -  Free radical biology & medicine  Sep 2009 
 - 548-58 p.  digital 
<br/><br/> Publication Type: Journal Article; Research Support, N.I.H., Extramural; Research Support, U.S. Gov't, Non-P.H.S. 
<br/><br/><label>ISSN: </label>1873-4596 
<br/><br/><label>Standard No.: </label>10.1016/j.freeradbiomed.2009.05.020  doi 
<br/><br/><label>Subjects--Topical Terms: </label><br/>Aldehydes--chemistry<br/>Apoptosis--drug effects<br/>Caspases--metabolism<br/>Cell Line<br/>Cholestanones--pharmacology<br/>Cholesterol--chemistry<br/>Cytochromes c--metabolism<br/>Dose-Response Relationship, Drug<br/>Drug Evaluation, Preclinical<br/>Humans<br/>Membrane Potential, Mitochondrial--drug effects<br/>Mitochondria, Heart--drug effects<br/>Models, Biological<br/>Myocytes, Cardiac--drug effects<br/>Oxidative Stress--drug effects<br/>Reactive Oxygen Species--pharmacology<br/>Receptors, Death Domain--metabolism<br/>Secosteroids--pharmacology<br/>Signal Transduction--drug effects